Week 2: Exploring Current OCD Research

Helga A L -

Hello, everyone!

 

Welcome back to my second blog post! I spent the majority of the past week conducting independent research, thoroughly reading several scientific papers published within the last five years discussing the current understanding of OCD and its treatment options. The papers I read comprehensively review many aspects of OCD and the available treatments; however, they can be condensed down into three major topics: comprehensive assessment and diagnosis of OCD, improving CBT and SSRI treatments, and changing understanding of OCD mechanisms. As the information is very dense in these papers, I will summarize them as best as I can.

 

Currently, studies have found that OCD is commonly under-diagnosed, comorbid with other mental health disorders, and unnoticed in patients for several years (Pampaloni et al., 2022). These aspects of the disorder make it difficult to diagnose OCD in patients, and due to it being a fairly complex disorder, a general guideline was proposed to include any and all possible factors that may influence OCD and aid in providing treatment. Pampaloni et al. (2022) recommended that these factors should include the patient’s OCD subtype, the level of insight into symptoms, the age of onset, and potential comorbidities. Other interesting aspects the authors recommended for complete assessment were neurocognitive and immunological factors as well as family history for OCD and other mental health disorders.

 

The last few factors stood out to me as I did not expect that OCD may be hereditary and influenced significantly by the immune system. However, a newer view in OCD neurobiology began to emerge that reveals that there are even more factors at play than previously speculated. Goodman et al. (2021) detail that many genes, molecular compounds, and brain region networks are likely involved in OCD. The authors proposed that specifically glutamate and serotonin transporters may have a role in OCD as indicated in initial studies. The review also emphasized that OCD may be the result of dysfunction in large brain networks, mentioning the cortico-striato-thalamo-cortical loop (CSTC) to be the most influential. Due to these findings, treatments such as TMS and DBS could be more precise and target regions involved in the CSTC loop.

 

CBT and SSRIs still remain as the main first-line treatments for OCD, but these treatments face many issues when utilized. Multiple studies have reported that CBT and SSRIs are only semi-effective for patients and that many would experience a relapse in symptoms after treatment ended. According to Nezgovorova et al. (2022), despite these treatments being considered first-line, many errors and gaps were noted in studies researching CBT, SSRIs, and combination treatments. The authors highlighted that many CBT trials were improperly conducted where participants had been undergoing SSRI treatment before taking part in the research studies and that the trials were completed over only a short-term period. Van Roessel et al. (2022) explored alternative medications and molecular compounds which could augment current SSRI treatment to increase its effectiveness. Many of the medications described affected serotonin, dopamine, and glutamate release in the brain, with various research trials providing supporting evidence.

 

These papers delved into the intricacies of OCD, its influences, and its treatments. There is an additional article I unfortunately did not have a chance to discuss here, but it will be included in my next blog post.

 

See you all then!

 

Pampaloni, I., Marriott, S., Pessina, E., Fisher, C., Govender, A., Mohamed, H., Chandler, A., Tyagi, H., Morris, L., & Pallanti, S. (2022). The global assessment of OCD. Comprehensive Psychiatry, 118, 152342. https://doi.org/10.1016/j.comppsych.2022.152342

Goodman, W. K., Storch, E. A., & Sheth, S. A. (2021). Harmonizing the neurobiology and treatment of Obsessive-Compulsive Disorder. The American Journal of Psychiatry, 178(1), 17–29. https://doi.org/10.1176/appi.ajp.2020.20111601

Nezgovorova, V., Reid, J., Fineberg, N. A., & Hollander, E. (2022). Optimizing first line treatments for adults with OCD. Comprehensive Psychiatry, 115, 152305. https://doi.org/10.1016/j.comppsych.2022.152305

Van Roessel, P. J., Grassi, G., Aboujaoude, E. N., Menchón, J. M., Ameringen, M. V., & Rodríguez, C. I. (2022). Treatment-resistant OCD: Pharmacotherapies in adults. Comprehensive Psychiatry, 120, 152352. https://doi.org/10.1016/j.comppsych.2022.152352

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Comments:

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    kelin_x
    Hi, Helga! OCD is such an interesting topic to do your senior project on! Thank you for giving us great summaries about the papers you read; I really appreciate that. My question for you is: as the main first-line treatments for OCD, how exactly do CBT and SSRIs work? Just wondering what makes those particular treatments first-line material compared to alternative treatments that don't hold as much significance. Looking forward to getting to know more about OCD's intricacies!
    Jolie L
    Hello! I was just wondering, since SSRIs work by preventing the reabsorption of serotonin into the brain, does that mean OCD is linked with low serotonin levels? This leads me to my main question: Can low serotonin levels affect an individual's ability to control their impulsive thoughts and actions? I've noticed that most obsessions and compulsions in OCD tend to be impulsive, so just wondering! I can't wait to read more about OCD :D!!
    Samee Saied
    Hi! Could you elaborate further on what other disorders OCD is mistaken for and how plausible it is to consistently identify these disorders from each other? And if OCD was to be misdiagnosed and given treatments for a different disorder would there be any adverse effects? Some physical problems can worsen if given the wrong treatment so I was wondering if that extends to these disorders. I’m eager to read the next update!
    Roberto D.
    Hello! You stated that the authors of the article proposed that serotonin and glutamate transporters are cornerstones in OCD. Do you believe this to be true? Do you believe other neurotransmitters like GABA, Dopamine, or Epinephrine have any effects on OCD? Thanks for the update!
    Ayesha Amirah
    Hello! In this update you stated how the main first-line treatments for OCD are only semi-effective, but I would like to know the overall time frames of the CBT and SSRIs treatments. It also brings me to wonder what type of withdrawal symptoms they might show after their treatment as well, and whether it is less frequent or gradually reverts back to its old recurrence. Thanks for this great update !
    helga_a_l
    Hello, Kelin! I appreciate your question. The exact mechanism and involvement of CBT and SSRIs in OCD is not 100% clear, but studies still refer to these treatments as first-line due to the larger amount of data collected and consistency seen in studies. CBT aims to help patients gain more awareness about the weight they place on their intrusive thoughts and includes Exposure and Response Prevention (ERP) techniques. This specific form requires the patient to experience a stimulus that is anxiety-inducing but prevents the patient from completing compulsions. As for SSRIs, they target serotonin receptors to inhibit their functions and increase serotonin in the brain. Hope this cleared up confusion!
    helga_a_l
    Hello, Jolie! Thank you for the question. The role of serotonin in OCD is still not completely clear unfortunately. I think it is possible there is a link between low serotonin levels and OCD as there are so many factors that influence the disorder. Coupled with evidence that OCD is often comorbid with other mental health disorders, lower levels may then influence an individual's responses to the obsessions and/or compulsions. There was some initial studies done that propose that serotonin systems in the brain are instead linked to another system that more directly impacts OCD, but more evidence is needed. I apologize for not being able to answer your question clearly, but hopefully research in the near-future will solve the mysteries surrounding OCD!
    helga_a_l
    Hello, Samee! Of course, I would love the elaborate! OCD is typically comorbid with a variety of other mental health disorders, and these can lead to misdiagnosis given the symptoms shown by the patient. Some of the comorbidities include anxiety disorders, Major Depressive Disorder, and Obsessive-Compulsive Related Disorders, which display similar symptoms but do not meet all the criteria for OCD such as ADHD and Body Dysmorphic Disorder. Unfortunately, these comorbidities can make it very difficult to differentiate between the disorders, leading to underdiagnosis of OCD in the population. Misdiagnosis and subsequent treatment may not always worsen symptoms, but it depends on the individual patient. Hope this helps answer your questions!
    helga_a_l
    Hello, Roberto! I personally agree with the studies, and I believe that glutamate, serotonin, and dopamine could have significant roles in OCD development and treatment. I have learned of many potential influences that could affect OCD, and I do not discount the possibility that multiple neurotransmitters are involved, and not just the three I mentioned above. The recent emphasis on brain networks in OCD studies lead me to believe this even more, given how interconnected the brain is. I think it would very interesting to find out if any other neurotransmitters are heavily involved in OCD as it would bring us one step closer to effectively treating it. Thank you for the question!
    helga_a_l
    Hello, Ayesha! The time frames for CBT and SSRIs from what I have seen mentioned in the scientific papers were up to 16 weeks for CBT treatment and around 8 to 12 weeks for SSRI treatment. These time frames were based off of how previous studies were conducted and the time it takes for a patient to respond to treatment. As for withdrawal symptoms, I believe it depends on the patient. OCD can differ significantly between individual patients and, as a result, returning symptoms after treatment differ as well. Thank you for your question!

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